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    IV型超敏反应
运行环境: Win9x/NT/2000/XP/2003 文件大小: 938 K
资源格式: Flash动画 资源等级: ★★★★
开 发 者: 网络 资源语言: 英文
所属分类: 人体免疫学 资源属性:     
下载次数: 808
资源添加: 审核:myworld21 录入:myworld21 添加时间: 2011-6-24
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解说词译文:

Harmful effects produced by the mechanisms of cell-mediated immunity are referred to as delayed hypersensitivity.

由细胞免疫途径造成的有害作用被称为迟发型超敏反应。

Delayed hypersensitivity is mediated by Th1 cells, also called TD cells, and is responsible for contact dermatitis, tissue damage in a variety of infectious disease, and rejection of tissue grafts.

迟发型超敏反应由Th1细胞介导,也被称为TD细胞,引起接触性皮炎,传染性疾病中的组织损伤和移植排斥反应。

A contact hypersensitivity such as poison oak dermatitis is initiated be contact with the antigen, which is also called the allergen.

接触性皮炎,如毒漆藤皮炎,是从接触抗原开始的,就是那个也被叫做变应原的东西。

During contact, a small molecule from the plant, called a hapten, binds to carrier protein in the host.

接触中,被叫做半抗原的,来自植物的一种小分子,与宿主的载体蛋白结合。

The carrier protein with bound hapten is ingested by a macrophage and the hapten-peptide is then presented on the surface of the macrophage on a Class II MHC.

与半抗原结合的载体蛋白被巨噬细胞吞噬,然后半抗原肽由MHC II类分子提呈于巨噬细胞的表面。

Th1 cells with T-cell receptors capable of recognizing the hapten-peptide antigen interact with the presented antigen, become activated and increase in numbers.

能够以TCR识别半抗原-肽抗原的Th1细胞与被提呈抗原相互作用,开始活化和增殖。

When the host comes in contact with the allergen a second time, the Th1 cells react with the hapten-peptide being presented on the macrophage and release cytokines resulting in attraction of more macrophages, followed by inflammation and skin lesions.

当宿主第二次与变应原接触时,Th1细胞与巨噬细胞提呈的半抗原肽反应,释放细胞因子,引起巨噬细胞趋化,接着是炎症和皮肤损伤。

Characteristic skin lesions appear after 24 hours, reaching their peak at 48-72 hours after this second exposure to the plant.

典型的皮肤损伤出现在再次接触那种植物24小时后,48-72小时后达高峰。


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